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山东省常见海产品蛋白质氨基酸含量分析 总被引：5，自引：2，他引：3
海产品蛋白质的营养价值及风味均与氨基酸含量有很大关系,为了更好地了解山东沿海地区蛋白质营养状况,根据山东沿海地区食物资源特点,作者采集了沿海地区常食用的２０余种海产品进行了蛋白质氨基酸分析,结果如下。１ 材料和方法１．１ 材料采自山东沿海地区新鲜海产品。１．２方法［１］１．２．１蛋白质的测定 以粗蛋白表示。以凯氏微量定氮法（Ｋｊｅｌｄａｈｌ法）测定总氮量,再乘一定的数值（转换系数为６．２５）,即为蛋白质质量。１．２．２氨基酸的测定 用氨基酸自动分析仪法完成。食物蛋白质经盐酸水解成为游离氨基酸后根… 相似文献
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目的：观察益气养阴汤辅助培美曲塞联合顺铂（PP）方案对中晚期肺腺癌气阴两虚证患者的临床疗效。方法：将60 例中晚期肺腺癌气阴两虚证患者随机分为治疗组和对照组,每组各30 例。对照组予以PP方案治疗,治疗组在对照组基础上加用益气养阴汤治疗,2组均治疗4个疗程。比较2组治疗前后的肿瘤标志物［癌胚抗原（CEA）、糖类抗原125（CA125）］、中医证候评分、Karnofsky功能状态评分（KPS）、癌因性疲乏量表（CFS）各维度得分情况及治疗后2组实体瘤近期疗效、药物毒副作用的发生情况。结果:疾病控制率治疗组为93.33%（28/30）,对照组为73.33%（22/30）,2组比较,差异有统计学意义（P<0.05）;2组CEA和CA125水平、中医证候评分和CFS评分均较治疗前下降,且治疗组低于对照组（均P<0.05）;2组KPS评分均较治疗前提高,且治疗组高于对照组（P<0.05）;治疗4个疗程后,治疗组在治疗过程中毒副作用的发生率较对照组低,但差异无统计学意义（P>0.05）。结论：益气养阴汤辅助PP化疗方案可以降低中晚期肺腺癌气阴两虚证患者CEA和CA125水平,在一定程度上缓解患者的临床不适症状,改善癌因性疲乏,提高生活质量,增效且不会增加药物的毒副作用。 相似文献
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山东省30种常见水产品油脂的脂肪酸含量分析 总被引：1，自引：0，他引：1
据刘玉军等１９８８年报道 ,近年来 ,长链不饱和脂肪酸受到广泛关注 ,水产品油脂中含有的各种不饱和脂肪酸对增进微血管壁的健全 ,防止心脑血管病的发生具有重要意义。为探讨鱼油资源的利用 ,本研究测定了山东地区常见的水产品中油脂提取物的脂肪酸构成及ｎ ３系列 (ＥＰＡ ＤＨＡ)的百分含量 ,旨在为新食物、药物资源的开发利用提供依据。１材料和方法１．１样品的采集青岛、烟台和济南等地区１．２样品处理鱼 :洗净去鳞 ,取鱼体一侧肌肉 ,制成肉糜。贝类及甲壳类 :洗净 ,去壳后取可食部分打碎１．３脂肪酸含量的测定［１］１．３．１脂肪… 相似文献
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通过细胞形态观察和CCK-8法研究HPN对HepG2细胞活力的影响。采用高糖高浓度胰岛素持续作用诱导Hep G2细胞建立胰岛素抵抗细胞模型,用多功能自动生化仪葡萄糖-己糖激酶法检测海普诺(HPN)对正常Hep G2细胞及胰岛素抵抗Hep G2细胞葡萄糖消耗量的影响。实验结果显示,低浓度的HPN对Hep G2细胞增殖没有影响。HPN与胰岛素协同作用Hep G2细胞,低浓度胰岛素对葡萄糖利用有一定的促进作用;当胰岛素浓度为1×10–6 mol/L时,HPN可明显促进Hep G2细胞葡萄糖消耗。进一步研究表明,HPN在5×10–8~1×10–7 mol/L浓度范围内可明显促进胰岛素抵抗Hep G2细胞的葡萄糖的消耗。HPN可明显改善Hep G2细胞胰岛素抵抗。 相似文献
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基于2009年7月9日姚安6.0级地震发生之前形变观测前兆异常,给出该地震三要素,总结其前兆异常特点,反思运用单台前兆观测资料预报地震过程中存在的问题,为今后的地震预测积累经验. 相似文献
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In this study, we isolated a polysaccharide from Branchiostoma belcheri (PBB) by enzymatic protein hydrolysis and alcohol precipitation. We investigated the effects of PBB supplementation on DNA
oxidation and growth of the transplanted tumor cells Sarcoma (S180) in mice. Sixty healthy Kunming mice weighing between 18 and 25 g were randomly assigned to 6 groups, each consisting of
10 animals. All the mice, except for the blank control group, were inoculated with S180 sarcoma cells into the axilla of the
left foreleg. PBB was given to mice by gavage at doses of 0 (model control), 25, 50, or 100 mg/kg b.w. in 0.2 ml saline for
30 days. The fifth group of S180-mice was given cytoxan (50 mg/kg) by peritoneal injection as a positive control group. The animals had free access to food
and water. The mice were sacrificed after the final treatment and blood was quickly collected. Spontaneous and oxidized DNA
damage of peripheral lymphocytes induced by H2O2 were analyzed by SCGE. O6-methyl-guanine (O6-MeG) was measured by high-performance capillary zone electrophoresis. The average tumor weights (0.856–1.118 g) of the three
PBB groups were significantly lower than that of the model control group (1.836 g) (p<0.05). The tumor inhibition ratios of the PBB groups were 39.1%–53.4% and similar to the cytoxan positive group (57.5%).
There were no significant differences in spontaneous DNA damage in peripheral lymphocytes among the groups. The oxidative
DNA damage induced by 10 μmol/L H2O2 in the 50 and 100 mg/kg b.w. groups were 246.1 AU and 221.7 AU, respectively, both of which were significantly lower than
that in the model group (289.0 AU; p<0.05). The plasma concentrations of O6-MeG in the 25, 50, and 100 mg/kg supplemented groups were 2.09 μmol/L, 1.86 μmol/L, and 1.63 μmol/L, respectively, all of
which were significantly lower than that of the model group (2.67 μmol/L; p<0.05). These results indicated that PBB may have antioxidative activity and thus reduce oxidation-induced DNA damage. 相似文献
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