故乡的原风景：膨胀与消失     

曾鸣 《湖南地质》2014,(7):85-86

今年春节前夕,我坐在大巴车上,从长沙向南疾驰两百公里,不用看表,也不用看地图,当车窗外天空的颜色慢慢变灰,公路两旁房子的外墙依次变黑,树叶渐渐看不出绿色……这就是我的故乡——冷水江的原风景。这座城市只比我们大十几岁,曾经迅速膨胀,现正迅速衰老。我一直担心,冷水江有一天会突然消失。直到我的双脚踏上了这片黑色的犹如肺痨病人脸色般的土地,心里才稍感踏实。至少,我的故乡还真实地存在于这个星球上。  相似文献   

安达市饮水型氟中毒与地质环境关系及开发利用建议     

李旭光  蔡贺  王长琪  郭常来 《地下水》2011,33(2):81-84

安达市是饮水型氟中毒高发区,分布面积大;控制安达市氟中毒的主要因素有地形地貌、地质、地球化学环境、水文地质及水化学环境等条件,通过对比分析氟含量与地质环境的关系,提出了饮水型氟中毒病区防病改水的建议.  相似文献   

吉林省双辽市氟中毒病区与非病区水文地球化学特征     

孙述海  迟宝明  孙亚红  熊绍礼 《吉林大学学报(地球科学版)》2011,(Z1):328-332,354

双辽市氟中毒病区与非病区地下水水化学类型均处于重碳酸型水带内。病区水文地球化学作用处于元素迁移—富集的过渡阶段或元素富集阶段的初期,其水化学类型呈现由HCO3→HCO3.Cl→Cl.HCO3型水的分带性展布,地下水溶解固体浓度为0.5～2.0 mg/L;非病区则处于元素迁移阶段,其水化学类型则为单一的HCO3.Ca型水,地下水溶解固体浓度一般均低于0.5 mg/L。地方性氟中毒的分布与区域水文地球化学特征之间存在着一定的联系,此种关系虽然不可作为病因看待,但仍可当作一种环境标志加以考虑。  相似文献   

麻痹性贝毒（PSP）在食物链的传递   总被引：2，自引：0，他引：2  

邹仁林 《南海研究与开发》1997,(2):1-8

扼要介绍织纺螺食物链中麻痹性贝毒（ＤＳＰ）的传递，因误食织纺引起民中毒事件。同时介绍什么是ＰＳＰ，ＰＳＰ的来源，全球ＰＳＰ中毒事件的记录、国内食织纹螺引发ＰＳＰ中毒事件及ＰＳＰ中毒症状和致死量。提醒喜食海鲜的人们提高警惕，在发生赤潮的前后时期尽量不吃或少食贝类海鲜，同时，呼呈医学部门和海洋环境部门积极加大投入、研制解药和保护好海洋环境，大力宣传ＰＳＰ对人们生命安全的威胁。增强科普知识，加强包含安全  相似文献   

日本鬼鲉蜇伤与P物质和生长抑素的关系及其粗毒对免疫细胞活性影响   总被引：1，自引：0，他引：1  

张克凌  刘晓萍  杜卫  于业军  孙衍增  王海青  秦士德 《海洋与湖沼》2000,31(4):378-383

１９９７年６月在山东省日照市岚山头附近海域收集日本鬼ｙｏｕ,采用放射凤学方法检测了注射毒腺粗提物大鼠局部肌肉组织、血浆、疹髓、延髓、中脑、下丘脑及免疫器官内Ｐ物质和生长抑素含量变化;用ＭＴＴ法检测了朱粗提物在整体和离体条件下对免疫细胞活性的影响;用生化方法检测了毒腺组织中透明质酸酶活性。结果表明,与对照组大鼠相比,实验组大鼠局部肌肉及延髓、中脑、胸腺中脾脏中Ｐ物质含量增加,而下丘脑中Ｐ物质含量下降  相似文献   

广东地方性氟中毒的类型,分布与防治对策     

周亮津 《广东地质》1993,8(2):21-27

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The genotoxic effects of benzo[a]pyrene and methamidophos on black porgy evaluated by comet assay     

刘日先洪华生  王新红王克坚 王春光 《中国海洋湖沼学报》2005,23(4):455-460

In this study, two common pollutants （benzo[a]pyrene and methamidophos） in marine environment were tested by comet assay for their inducement of in vivo genotoxic effect to the blood cells of black porgy （ Acanthopagrus schlegeli）. The fish was exposed to 2 μg/L of benzo[a]pyrene （BaP） and methamidophos, and their mixture. The assay was performed on whole blood at 2 h, 5 h, 24 h and 96 h exposure intervals. A significant increase in DNA damage was observed in each treatment with the pollutants. Additive effect of BaP and methamidophos was also found in the experiment. However, the decrease ratios of DNA damage for 5 h and 96 h exposure interals compared with 2 h and 24 h exposure ones, respectively, were noticed. This phenomenon may be explained by the function of repairing process via enzyme cytochrome P450 in the animal. Evidence of the genotoxicity of organophosphorus pesticides （OPs） and polynuclear aromatic hydrocarbons （PAHs） on marine fish are discussed in this paper.  相似文献   

河套地区盐碱化和砷氟中毒问题探讨   总被引：3，自引：0，他引：3  

王喜宽黄增芳  赵锁志苏美霞  李世宝张青 王忠 《岩矿测试》2007,26(4):328-330

内蒙古河套地区多目标地球化学调查表明，河套地区总体上为无公害、绿色土地。作为我国北方重要的粮食生产基地可以为全国生产出优质的绿色产品；但河套地区也具有比较严重的生态地球化学问题，首先是土壤的盐渍化制约了河套地区农业的发展，其次是全区以砷氟中毒为主的地方病比较严重，极大地影响了该区人民的身体健康。此外，包钢尾矿坝作为一个局部性的生态问题，对包头市民产生重大的影响，对其周围的居民身体健康造成严重危害。  相似文献   

A study of fluoride occurrence in groundwater and dental fluorosis in Nathenje, Lilongwe, Malawi     

《中国地球化学学报》2006,25(B08):60-60

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Fluorosis and arsenicosis： An update     

Lakshmi Narasimharao Kodali Jagadiswararao Ramayanam 《中国地球化学学报》2006,25(B08):65-65

Endemic fluorosis, caused by excess fluoride in water, is widespread in the arid and semiarid tracts of India. This disorder is of recent origin and developed due to certain faulty water management practices. After Eager reported ‘dental mottling＇ among emigrants from Pozzuli in Italy in 1901, several such reports came from all over the world. From 3 works published independently from Pennsylvania, Arizona and South Africa in 1931, it was known that this disorder is caused by drinking water containing over 1 mg/L fluoride during formation of permanent teeth among children.‘Skeletal fluorosis/Osteofluorosis＇, affecting bones and joints among adults was reported by Moeller and Gudjonsson in 1932 due to accumulation of fluoride in hard tissues by drinking water with over 7 mg/L fluoride for several years. In course of time fluorosis has been reported in over 22 countries from all over the world. Fluorosis spreads now among 67 million people in the entire arid and semiarid tracts of India. The work of National Institute of Nutrition of India at Hyderabad in 1977 revealed the spread of endemic ‘genu valgum＇ （knock-knee） crippling lower limbs right from childhood in association with fluorosis in most parts of India. Although preliminary work revealed that genu valgum is caused by drinking waters poor in calcium and rich in molybdenum, efforts these disorders by supplying low-fluoride water should be taken up to know its exact cause to the communities could not so eradicate The governmental efforts to eradicate the problem.  相似文献