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A critical test of a general circulation model is its performance on the regional scale. In this paper we examine the summer climatology of the CSIRO4 (4-layer) climate model over the Australian tropical region. The benchmark for the study is the positioning of the monsoon equatorial trough. We compare the CSIRO4 model climatology with the climatologies from the GFDL and GISS models and we report on the sensitivity of the position of the monsoon shear line and the strength of the monsoon westerly winds to the doubling of carbon dioxide in the atmosphere. The model results show that under the greenhouse scenario the monsoon is strengthened, but the average location of the monsoon shear line is not sensitive to the doubling of CO2. Offprint requests to: BF Ryan  相似文献   
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Chronic exposure to organic contaminants such as polychlorinated biphenyls (PCBs) can lead to the development of resistance to these chemicals, a condition associated with reduced response of CYP1A1, a pollutant-inducible biomarker. We measured CYP1A activity (ethoxyresorufin o-deethylase, EROD) and PCB concentrations in feral fish from the Town Branch/Mud River system (Logan County, KY), a stream historically contaminated with PCBs and partially remediated. As a first step in evaluating the possible development of resistant populations in this system, we measured CYP1A expression and PCB body burdens in resident fish from sites we previously characterized as containing biologically significant levels of CYP1A inducing compounds. Mean PCB concentrations in edible flesh ranged from 75.2 to 16.7 microg/g in fish collected from Town Branch remediated sites and were relatively low (1.23 microg/g) in Town Branch reference site fish. However, hepatic CYP1A activity was similar among individuals of most species collected from reference and contaminated/remediated sites. The absence of elevated CYP1A levels in resident fish species despite the presence of significant PCB body burdens may indicate these fish have developed reduced sensitivity to CYP1A induction, a condition associated with acquired resistance to toxicants.  相似文献   
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